Cultureplaces

Go to: Abstract The feminist movement purports to improve conditions for women, and yet only a minority of women in modern societies self-identify as feminists. This is known as the feminist paradox. It has been suggested that feminists exhibit both physiological and psychological characteristics associated with heightened masculinization, which may predispose women for heightened competitiveness, sex-atypical behaviors, and belief in the interchangeability of sex roles. If feminist activists, i.e., those that manufacture the public image of feminism, are indeed masculinized relative to women in general, this might explain why the views and preferences of these two groups are at variance with each other. We measured the 2D:4D digit ratios (collected from both hands) and a personality trait known as dominance (measured with the Directiveness scale) in a sample of women attending a feminist conference. The sample exhibited significantly more masculine 2D:4D and higher dominance

What Can’t Be Debated on Campus Pilloried for her politically incorrect views, University of Pennsylvania law professor Amy Wax asks if it’s still possible to have substantive arguments about divisive issues. ILLUSTRATION:  JOHN CUNEO By Amy Wax Feb. 16, 2018 9:51 a.m. ET 824  COMMENTS There is a lot of abstract talk these days on American college campuses about free speech and the values of free inquiry, with lip service paid to expansive notions of free expression and the marketplace of ideas. What I’ve learned through my recent experience of writing a controversial op-ed is that most of this talk is not worth much. It is only when people are confronted with speech they don’t like that we see whether these abstractions are real to them. The op-ed, which I co-authored with Larry Alexander of the University of San Diego Law School, appeared in the Philadelphia Inquirer on Aug. 9 under the headline,  “Paying the Price for the Breakdown of the

DNA-based scores are getting better at predicting intelligence, risks for common diseases, and more.  by  Antonio Regalado   February 21, 2018 hen Amit Khera explains how he predicts disease, the young cardiologist’s hands touch the air, arranging imaginary columns of people: 30,000 who have suffered heart attacks here, 100,000 healthy controls there. There’s never been data available on as many people’s genes as there is today. And that wealth of information is allowing researchers to guess at any person’s chance of getting common diseases like diabetes, arthritis, clogged arteries, and depression. Doctors already test for rare, deadly mutations in individual genes. Think of the  BRCA  breast cancer gene. Or the one-letter mutation that causes sickle-cell anemia. But such one-to-one connections between a mutation and a disease—“the gene for X”—aren’t seen in most common ailments. Instead, these have complex causes, which until recently have remained